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amounted to 200 pounds per person, 22 pounds above the level in 197024 Even though Americans have been gradually switching to less fatty meats, they lose the bene t by consuming more calories and less nutritious foods Indeed, the increase in caloric intake in the United States is linked to consuming more processed foods Back in the early 1970s during the Nixon administration, the United States faced a problem that resurfaced in 2007 and 2008: rising food prices In 1973, food prices hit an annual in ation level of 8 percent President Nixon devised a domestic food policy to help ease the impact on American wallets Nixon held down the prices of processed foods while allowing the prices of raw farm produce to rise25 Hence, processed foods became drastically less expensive for the average American consumer than unprocessed foods In order to support the processing and help his approval ratings, the Nixon administration began to heavily subsidize corn, a legacy still with us today As one researcher discovered, 13 types of processed corn are found in a typical McDonald s meal Additionally, corn and its by-products, such as high fructose corn syrup, make up about 25 percent of all of the edible items available on our supermarket shelves26 High fructose corn syrup permeates nearly every possible type of processed food It s harder for your body to digest and results in the consumption of higher than normal levels of fructose Higher levels of fructose, in turn, lead to increased levels of type II diabetes and surging rates of obesity27 The dependence on corn feed and increased intake of processed foods, calories, and protein also spill over into the environmental realm: The average American diet produces an extra ton and a half of carbon dioxide-equivalent annually (in the form of actual carbon dioxide as well as methane and other greenhouse gases) compared to a strictly vegetarian diet By cutting down on just a few eggs or hamburgers each week, greenhouse gas emissions would be drastically reduced28 In 2002, energy used for food production accounted for 17 percent of all fossil fuel use in the United States29 In 2007, another period of food price in ation kicked off, and as prices soared, questions about the way we produce and consume food became crucial to matters of security, trade, poverty, and environment, not to mention health Demand for food has been on the rise both in terms of the demand for human consumption and for ethanol.

18 the prostate About 80% of DHT is produced in peripheral tissues and the remaining 20% is secreted directly by the testis Furthermore, approximately 30 40 g of estradiol is produced by the adult male, mainly in peripheral tissues, such as adipose tissue, bone, prostate, and brain Insight into the biologic actions of estradiol in the male is rather recent Estrogens have an important effect on the nal phases of skeletal maturation and bone mineralization in puberty In addition, from some studies in elderly men it appears that estrogen levels show a higher correlation with bone mineral density (BMD) than androgen levels [3] Impaired estrogen action in men leads to dyslipidemia and to endothelial dysfunction Observation in men with aromatase de ciency is linked to a complex dysmetabolic syndrome characterized by insulin resistance, diabetes mellitus type 2, acanthosis nigricans, steatosis hepatis, and signs of precocious atherogenesis, remedied by estrogen administration Estrogen effects on the brain are also increasingly recognized [4] Though the effects of estrogens in the male are undeniably biologically signi cant, estrogen de ciency as a clinical entity is sporadic in men Since T is a precursor molecule for estradiol, it is usually associated with (severe) androgen de ciency

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de ciency of the testicular secretion of androgens mainly needed for virilization, anabolic effects, and expression of male sexual behavior If the cause of hypogonadism lies in the testis itself, this is referred to as primary or hypergonadotropic hypogonadism Hypothalamic or pituitary disorders of testicular dysfunction are grouped together under the term secondary or hypogonadotropic hypogonadism [1] Late onset hypogonadism (LOH) is the term recommended by the International Society for the Study of the Aging Male (ISSAM), the International Society of Andrology (ISA), and the European Association of Urology (EAU), to replace the previous terminology of andropause, androgen de ciency of the aging male (ADAM), and partial androgen de ciency of the aging male (PADAM) [2] It is a clinical and biochemical syndrome associated with advancing age and is characterized by typical symptoms and a de ciency in serum T levels It may result in signi cant detriment in the quality of life and adversely affect the function of multiple organ systems

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